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. Coupled with the altered expression of various genes that control bone marrow mobilization in the two classical Nur77-KO and full Nur77-KO mice in comparison with WT mice, we speculate the bone marrow, liver, and spleen phenotypes noticed exclusively in classical Nur77-KO mice are caused by an imbalance amongst the genomic motion of Nur77 being a transcription element vs .
). It is actually consequently extremely not likely that altered differentiation of Ly6Clo monocytes is the cause of the spleen and liver abnormalities noticed completely in classical Nur77-KO mice.
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We had been intrigued by the phenotypic distinctions involving aged classical Nur77-KO and comprehensive Nur77-KO mice and determined to investigate a doable trigger for this discrepancy in more depth. Classical Nur77-KO mice were originally generated by insertion of the NeoR cassette in exon two from the Nr4a1
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